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The aim of the study is to test the dynamics of ischemic damage after bilateral internal carotid artery occlusion and to examine the impact of Triamcinolone (Glucocorticoid) and Lornoxicam (NSAID) on the general condition of the eye and retina. Ischemia takes part in almost all vitreoritenal diseases and can be caused by circulatory deficiency as a result of diabetic or hypertonic angioretinapathy, carotid or ophthalmic artery stenosis, and trauma. Eye ischemia frequently leads to loss of vision. As the result of development of hypoxic eye damage signaling pathways are activated which start up growth processes and therefore ischemia can be the cause of proliferative forms of the disease.